MedicalResearch.com Interview with:
Bradley C. Clark, MD
Pediatric Cardiology Fellow – 3
rd Year
Division of Cardiology
Children's National Health System
Washington, DC 20010
Medical Research: What is the background for this study? What are the main findings?
Dr. Clark: After consulting on multiple pediatric emergency room patients with K2 (synthetic cannabinoid) ingestion and electrocardiogram (ECG) abnormalities, my co-authors and I decided that it was worth taking a more detailed look at the potential cardiac effects of synthetic
cannabinoids.
We did a retrospective chart review and discovered a total of 8 patients in a 3 year period (2011 – 2014) at our institution with reported synthetic cannabinoid ingestion and concern for myocardial injury. There were 3 individuals with evidence of ECG abnormalities in a segmental pattern with increased cardiac enzyme levels (troponins). The other 5 individuals had ECG abnormalities either without troponin elevations or were not specifically tested. Each individual that had an echocardiogram performed had normal intracardiac anatomy with normal biventricular systolic function.
Given the elevated troponin levels and ECG abnormalities, there was a suspicion for myocardial ischemia in this small subset of patients without meeting specific criteria for myocardial infarction. Interestingly, these individuals had completely normal echocardiograms and had no other potential cause of myocardial ischemia discovered by history. Additionally, these were all teenage pediatric patients with documented K2 exposure without evidence of exposure to illegal substances.
K2 and other synthetic cannabinoids are known to cause analgesia and euphoria and can lead to a lack of symptomatology. Therefore, individuals with synthetic cannabinoid ingestion may not complain of the prototypical cardiac symptoms (chest pain, shortness of breath, palpitations) and may not have the workup to diagnose potential myocardial ischemia.
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